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[1] [2] Unlike phentermine's central nervous system stimulation, tirzepatide works through multiple mechanisms: it enhances insulin secretion in a glucose-dependent manner, suppresses glucagon release, slows gastric emptying, and reduces appetite through central pathways

Cellular uptake is facilitated by the H-coupled oligopeptide transporter PepT1, through which KPV accumulates intracellularly and suppresses downstream pro-inflammatory cytokine expression, including TNF-, IL-6, and IL-8 (Dalmasso et al

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The synergistic approach may accelerate results in priority areas while the injectable component addresses underlying causes of skin laxity systemically